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By Hans-Joachim Priebe; Karl Skarvan

ISBN-10: 0585383774

ISBN-13: 9780585383774

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Circulation 1993;87:1806–15. 31 Fabiato A. Calcium induced release of calcium from the cardiac sarcoplasmic reticulum. Am J Physiol 1983;245:C1-14. 32 McCormack JG, Halestrap AP, Denton RM. Role of calcium ions in reperfusion of mammalian intramitochondrial metabolism. Physiol Rev 1990;70:391–425. 33 Lehninger AL, Reynafarie B, Vercesi A. Transport and accumulation of calcium in mitochondria. Ann NYAcad Sci 1978;307:160–76. 34 Denton RM, McCormack JG. On the role of the calcium transport cycle in the heart and other mammalian mitochondria.

A decrease in resistance or an increase in aortic compliance will decrease the amplitude of reflected waves and moduli oscillation, whereas an increase in resistance or a decrease in aortic compliance will have the opoposite effect. 34 The Page 41 overall contribution of the frequency dependent oscillatory impedance to flow in the normal systemic circulation is, however, only approximately 10% of the total external load. As a result of anatomical differences, it is significantly higher in the pulmonary vascular bed.

14 A failing ventricle is deprived of these compensatory mechanisms and, consequently, becomes exquisitely sensitive to any increase in afterload. Contractility The third major determinant of ventricular function is the contractile state of the myocardium. A change in myocardial contractility will breech the confines of the diastolic and end systolic P/V relationship. With an increase in contractility, the end systolic points of the P/V loop will shift upwards and to the left of the original active end systolic P/V relationship; consequently, stroke volume will increase despite unchanged preload and afterload.

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Cardiovascular physiology by Hans-Joachim Priebe; Karl Skarvan

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