By John F. Van Vleet, Victor J. Ferrans (auth.), Thomas Carlyle Jones D.V.M., D.Sc., Ulrich Mohr M.D., Ronald Duncan Hunt D.V.M. (eds.)
This quantity is one in every of a collection of concise, good illustrated and authoritative reports of typical and experimental lesions of laboratory animals (rat, mouse and hamster) prepared via organ platforms. designated differential diagnostic standards and terminology are given emphasis. incorporated are valuable info on pathogenesis, etiology, biologic habit, frequency in numerous traces and lower than various laboratory stipulations.
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Extra info for Cardiovascular and Musculoskeletal Systems
Incomplete regenerative processes and compensatory hypertrophy of unaffected fibers are followed by severe dilation of the ventricles in the final phase (Doerr and Mall 1979). Consistent chronic pulmonary and hepatic congestion allow the study of secondary effects and their modification by drugs (Desjardins et al. 1989; Jasmin and Proschek 1983; Rossner and Sachs 1978; Rossner and Coudrai 1986). The frequent thrombosis (Fig. 19) in advanced cases of congestive heart failure in hamsters (Desjardins 21 and Mueller 1988) is comparable to observations in man (Sandritter 1981 a, b) and offers further areas for comparative study.
Recent Adv Stud Cardiac Struct Metab 2: 423--432 dose-dependent perturbations of lipid mem- Myocardial Disease Associated with the Feeding of Unsaturated Vegetable Oil, Rat Grasso P, Mujr A, Goldberg F, Batsone E (1968) Studies on Brown FK. IV. Cytopathic effects of Brown FK on cardiac and skeletal muscle in the rat. Food Cosmet Toxicol6: 13-24 Griffing WJ, Todd GC, Pierce EC (1970) Ultrastructural changes in rat skeletal and cardiac muscle induced by A 204. Antimicrob Agents Chemother 366-373 Hagopian M, Gershon MD, Nunez EA (1972) An ultrastructural study of the effect of reserpine on ventricular cardiac muscle in active and hibernating bats (Myotis lucifugus).
Rats killed after 36 h have enlarged and vacuolated mitochrondria with electronlucent matrices and reduced cristae (Fig. 27). Swollen mitochrondria are seen also in rats killed at 72-120 h following a single oral dose of 149 mg monensin sodium/kg body wt (Mollenhauer et al. 1981, 1984). The outer mitochondrial membranes and other cellular organelles remain intact. Differential Diagnoses The myocardial damage produced in rats exposed to toxic levels and ionophores is nonspecific with respect to light microscopic and ultrastructural features.
Cardiovascular and Musculoskeletal Systems by John F. Van Vleet, Victor J. Ferrans (auth.), Thomas Carlyle Jones D.V.M., D.Sc., Ulrich Mohr M.D., Ronald Duncan Hunt D.V.M. (eds.)