By Comité de Trauma del Colegio Americano de Cirujanos
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22 ABC of Kidney Disease Coresh J, Astor BC, Greene T, Eknoyan G, Levey AS (2003) Prevalence of chronic kidney disease and decreased kidney function in the adult US population: Third National Health and Nutrition Examination Survey. Am J Kidney Dis 41: 1–12. Go AS, Chertow GM, Fan D, McCulloch CE, Hsu CY (2004) Chronic kidney disease and the risks of death, cardiovascular events, and hospitalization. N Engl J Med 351: 1296–305. Halbesma N, Jansen DF, Heymans MW et al. (2011) PREVEND Study Group: Development and validation of a general population renal risk score.
Ix) CKD-Mineral and Bone Disorder (CKD-MBD) See Chapter 4. (x) Acidosis and hyperkalaemia See Chapter 4. (xi) Manage ﬂuid overload Manage ﬂuid overload while avoiding deterioration in renal function from diuretic use. See Chapter 4. (xii) Dialysis See Chapter 10. (xiii) Palliative care See Chapter 9. 21 treatment. There will be a large group of patients, particularly in Stage 3A CKD, who will not experience signiﬁcant deterioration in renal function but may suffer disproportionate anxiety about their health without adequate explanation.
In many cases, this led to iron overload, infections, allergic reactions and increased sensitization to human leukocyte antigens with consequential difﬁculties if transplantation was considered. The management of anaemia related to CKD was revolutionized by the introduction of recombinant human erythropoietin in the late 1980s (National Clinical Guideline Centre 2011). Since then, longer-acting erythropoietin analogues have become available, and this class of drugs is known as the erythropoiesis-stimulating agents (ESAs), which have allowed earlier and more sustained treatment of the anaemia.
ATLS by Comité de Trauma del Colegio Americano de Cirujanos